Central to inflammatory bowel disease (IBD) pathogenesis is lack of mucosal barrier function. deletion on vascular endothelial cells (research with knockdown in intestinal epithelial ethnicities or pharmacologic research highlighted Adora2b-driven phosphorylation of vasodilator-stimulated phosphoprotein (VASP) as a particular hurdle repair Lepr response. Likewise research in hereditary mouse versions or treatment research with an Adora2b agonist… Continue reading Central to inflammatory bowel disease (IBD) pathogenesis is lack of mucosal