If highly pathogenic H5N1 influenza infections acquire affinity for human being instead of avian respiratory epithelium, will their susceptibility to neuraminidase (NA) inhibitors (the likely 1st line of protection against an influenza pandemic) switch aswell? Adequate pandemic preparedness needs that this query be clarified. oseltamivir. Twenty infections were genetically steady. The triple N158S/Q226L/N248D HA mutation… Continue reading If highly pathogenic H5N1 influenza infections acquire affinity for human being