Atherosclerotic plaque formation is usually fueled from the persistence of lipid-laden macrophages in the artery wall. of CD68 was unchanged (Fig. 1c). Related results were observed in hypercholesterolemic data oxLDL but not LDL improved macrophage manifestation of and mRNA (Fig. 1d Supplementary Fig. 1e). Immunoblot analysis confirmed improved cell-associated netrin-1 protein in oxLDL-treated macrophages (Fig.… Continue reading Atherosclerotic plaque formation is usually fueled from the persistence of lipid-laden